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These unstable molecules are produced during normal metabolism and through exposure to environmental factors such as pollution, ultraviolet light and physical stress

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Our results support a causal pathogenic axis in PND: surgical stress triggers STAT1-mediated transcriptional upregulation of GLS1 in activated microglia, which drives excessive extracellular glutamate accumulation, competitive inhibition of neuronal system Xc - , and iron-dependent lipid peroxidation, ultimately leading to neuronal ferroptosis, synaptic damage, and cognitive impairment